Читать книгу Neurosyphilis. Modern Systematic Diagnosis and Treatment Presented in One Hundred and Thirty-Seven Case Histories онлайн

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PERIPHERAL NERVE PALSIES

TEMPORARILY FROM COMPRESSION BY TOURNIQUET

FRIEDREICH’S ATAXIA

SUBACUTE COMBINED DEGENERATION OF POSTERIOR AND LATERAL COLUMNS

Posterior column disease

FOCAL LESION IN GRAY MATTER OF CORD

INFANTILE PARALYSIS (ACUTE ANTERIOR POLIOMYELITIS)

PROGRESSIVE MUSCULAR ATROPHY

(chronic anterior poliomyelitis)

Anterior cornua of cord

AMYOTROPHIC LATERAL SCLEROSIS

SYRINGOMYELIA

THROMBOSIS OF ANTERIOR SPINAL ARTERY

LANDRY’S PARALYSIS

Anterior cornua and peripheral motor nerves

MYOPATHIES

(pseudohypertrophic and atrophic types)

MuscLe itself

AMYOTONIA CONGENITA

FAMILY PERIODIC PARALYSIS

(during attacks)

INCREASED INTRACRANIAL PRESSURE

(especially hydrocephalus and tumors of posterior fossa)

PNEUMONIA

IMMEDIATELY AFTER ATTACK OF MAJOR EPILEPSY

(post-epileptic coma)

TOXIC COMA

(uremia, morphine, etc.)

DURING SPINAL ANESTHESIA

COMPLETE TRANSVERSE LESION OF CORD

Purves Stuart

Chart 16

1. What is the cause of the negative serum W. R.? It is claimed that 3 to 5% of all cases of general paresis yield a negative blood serum. In this particular case, there had been considerable treatment, including some Swift-Ellis treatment, so that it may be that this treatment had reduced a formerly positive blood serum W. R. to a negative one.